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Figure 1 (a) Thin section of an inclusion-bearing Lucke tumor cell with typical herpesvirus particles in various stages of development in the nucleus. Magnification x19400. (b) Enveloped extracellular virions. Magnification x24000. (c) Negatively stained nonenveloped particles showing typical herpesvirus morphology. Magnification x 110 000. Reproduced from Granoff A (1972) Lucke tumor-associated viruses - a review. In: Biggs PM, de-The G, and Payne LN (eds.) Oncogenesis and Herpesviruses, pp. 171-182. Lyons: International Agency for Research on Cancer, with permission from WHO.

Figure 1 (a) Thin section of an inclusion-bearing Lucke tumor cell with typical herpesvirus particles in various stages of development in the nucleus. Magnification x19400. (b) Enveloped extracellular virions. Magnification x24000. (c) Negatively stained nonenveloped particles showing typical herpesvirus morphology. Magnification x 110 000. Reproduced from Granoff A (1972) Lucke tumor-associated viruses - a review. In: Biggs PM, de-The G, and Payne LN (eds.) Oncogenesis and Herpesviruses, pp. 171-182. Lyons: International Agency for Research on Cancer, with permission from WHO.

studies, this agent (KHV) was also known as carp interstitial nephritis and gill necrosis virus. Consideration of developmental, morphological, and, most recently, genetic attributes have led to the virus being proposed formally as a member of the proposed family Alloherpesviridae. The complete genome sequences of three strains of KHV (from Japan, USA, and Israel) were published in 2007.

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